Wilson's ilness is an autosomal recessive disrupt that manifests itself in later childhood or in adulthood with hepatic, neurological, psychiatric findings or a combination of the above. Onset with neurological involvement is extremely rare after age 50. Neurological findings are present as the initial clinical manifestation in 40-60% of affected individuals. Wilson's disease should be investigated in any child with liver disease of unknown etiology, especially if there is a consanguineous marriage. Early diagnosis, especially in children, can prevent the disease from progressing, at least in the first place, and allow them to live a normal life. Pediatric patients require chelation or zinc therapy throughout their lives, along with a low-copper diet. Free radicals with antioxidants, which have been the most studied and researched topics recently, have gained more importance over time. When everything is normal; when the living metabolism is healthy, antioxidants with free radicals are in stabilize. However, when the stabilize shifts in favor of free radicals, a tendency to illness caused by oxidative stress is observed. In particular, the rise in exogenous free radical sources such as environmental pollution, alcohol and cigarette consumption, forest burning, and X-rays and UV radiation can damage carbohydrates, fats, proteins, and DNA in the human body, leading to oxidation. Oxidative stress can be combated by increasing antioxidant capacity. This study examines the role of oxidative stress in wilson's disease..